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Since SARS-CoV-2 activates mitochondrial ROS-mediated feedback loops that produce long-term changes in the redox status and endothelial function of the host, leading to cardiovascular disease and lung injury (Chang et al., 2021), and endothelial cells are key players in inflammatory pathologies, such as acute respiratory distress syndrome, thrombosis, and atherosclerosis

[9] Tittmann K, Wille G, Golbik R, Weidner A, Ghisla S, Hubner G

6 The G6PD-deficient variants are grouped into different classes corresponding with disease severity( Table 1 1,7 )

The lipotoxicity may exacerbate mitochondrial dysfunction and promote the production of ROS [61], which can enhance inflammation, thereby impairing angiogenesis and adversely affecting placental structure and function [62]

[DOI] [PubMed] [Google Scholar] 13.Hunter EA, Grimble RF